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Froman, G. , Signas. C, Nilson, B. , and Lundberg, M. EMBO 6, 2351-2357,1987 Froman, G, Switalski, LM, Speziale, P Hook, M. J. BioI. Chem. M, and Colvin, R B Lab. Invest 45, 120-129,1981 Fyrand,O Br J. H. 1. S. J. BioI. Chem. R. N. Eng. J. Med. , Knudsen, K, Damsky, C, and Comoglio, PM. Exp. Cell. Res. , and Delano, E. , and Pearlstein, E Proc. Natl. Acad. Sci. 1. J. Cell. Bioch. , and Minter, D. , and Burgess, L. J. Invest. Dermatol. , F. J Cell. Biochem. M. Proc. Natl. Acad. Sci. M. Cell 18, 1043-1051, 1979b Hayashi, M.
In all the further steps of the woundhealing process fibronectin appears somehow to be involved (Grinnell, 1984). , 1981). Following hemostasis, invasion of the wound region by neutrophils and monocytes, which control infection and remove tissue debris, occurs. , 1985). Neutrophils can also proteolytically modify fibronectin. , 1983) fibronectin allows neutrophils to enhance their own adherence to endothelial cells. The ingrowth of fibroblasts and endothelial cells from the subdermis and the formation of granulation tissue depends again on the presence of fibronectin.
Hulser In vitro they are closed. the gap junctions were 36 Merten If I suppose for instance that they are open, they could be very interesting for the aggregate cultures because you make aggregates normally, you have some problems in the center of the aggregates. you have open gap junctions, the products not if nutritional But if perhaps there could be diffusion between the cells of but within the cells Yes, there are some in an animal or in an organ but these too, to the center of the aggregate.