Chronic Kidney Disease and Renal Transplantation by M. Sahay

By M. Sahay

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Journal of the American Society of Nephrology, Vol. 21, No. 2, (February 2010), pp. 237-248, ISSN 1046-6673. , & Esbrit, P. (2008). Parathyroid hormone-related protein interacts with vascular endothelial growth factor to promote fibrogenesis in the obstructed mouse kidney. American Journal of Physiology. Renal Physiology, Vol. 295, No. 2, (June 2008), pp. F415-F425, ISSN 03636127. V. (2002). Apoptotic pathways of oxidative damage to renal tubular epithelial cells. Antioxidant & Redox Signaling, Vol.

References [1] Coresh J, Selvin E, Stevens LA, Manzi J, Kusek JW, Eggers P, Van Lente F, Levey AS: Prevalence of chronic kidney disease in the United States. JAMA 2007; 298: 2038-47 [2] Stojiljkovic L, Behnia R: Role of renin angiotensin system inhibitors in cardiovascular and renal protection: a lesson from clinical trials. Curr Pharm Des 2007; 13: 133545 [3] Ganne S, Arora S, Dotsenko O, McFarlane S, Whaley-Connell A: Hypertension in people with diabetes and the metabolic syndrome: Pathophysiologic insights and therapeutic update.

2003). , 1994). , 2008). In this study, PTHrP was found to be upregulated, even in PTHrPoverexpressing mice. , 2008), PTH1R was not downregulated after ureteral obstruction in mice. , 2006). Furthermore, our recent in vitro findings indicate that PTHrP upregulates several proinflammatory factors in tubuloepithelial cells and promotes monocyte/macrophage migration. Moreover, ERK mediated NF-kB activation appears to be an important mechanism whereby PTHrP triggers renal inflammation. , 2008). Finally, since sustained renal inflammation is closely related to fibrogenesis, these data point to PTHrP as a likely pro-inflammatory and pro-fibrogenic cytokine in the damaged kidney.

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